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SKU: 86698506773
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doi: 10.5435/JAAOSGlobal-D-25- 6: Mayfield CK, Bolia IK, Feingold CL, Lin EH, Liu JN, Rick Hatch GF, Gamradt SC, Weber AE

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We hypothesize that ArfGAP1 could mediate LRRK2-induced toxicity either by i) serving upstream as a GAP-like protein to enhance the GTPase activity of LRRK2, or ii) serving downstream as a substrate of LRRK2-dependent phosphorylation