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Working hypothesis in the PPPM framework of COVID-19-caused neurological complications We hypothesize a series of molecular events and signaling pathway alterations occur in the pathophysiological processes of COVID-19-caused neurological complications

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Oxidative stress is tied not only to poor sleep but also to chronic conditions such as heart disease, neurological problems, and inflammation

NNMT is highly expressed in adipose tissue and plays a role in cellular methyldonor economy and nicotinamide processing