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Intragastric ascorbic but not uric acid is depleted in relation with the increased pH in patients with atrophic body gastritis and H

10.1212/01.wnl.0000095214.53646.72 242 SealeP.ConroeH

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Water (Aqua), Glycerin, Dipropylene Glycol, Niacinamide, Ethanol, Lecithin, Hydrogenated Lecithin, Saccharomyces Ferment Filtrate, Ethylhexylglycerin, Allantoin, Tocopherol, Tocopheryl Acetate, Arginine, Arbutin, Carbomer, Trehalose, Dextrin, Sea Water, Milk Extract (1ppm), Linolenic Acid, Stearic Acid, Ascorbyl Palmitate, Linoleic Acid, Panthenol, Phytosphingosine, Hyaluronic Acid, Adenosine, Palmitoyl Pentapeptide-4, 1,2-Hexanediol, PEG-40 Hydrogenated Castor Oil, Ethylhexyl Stearate, Disodium EDTA, Sorbitan Isostearate, Polysorbate 60, Glutathione, Diisostearyl Malate, PEG-60 Hydrogenated Castor Oil, Bis-PEG-18 Methyl Ether Dimethyl Silane, PEG/PPG-26/26 Dimethicone, PEG-11 Methyl Ether Dimethicone, Tromethamine

At 12 h, hepatic d-lactate is 13 % lower and GLO activity is 52 % lower following BSO, which is fully restored by the exogenous addition of GSH [76]