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Such toxic lipid intermediates activate protein kinases such as PKC, inflammatory pathways including JNK/NF-B, and toll-like receptors (TLRs), thereby amplifying sterile immune responses [36]

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KD can mitigate the metabolic reprogramming and systemic inflammation in SARS-CoV-2 infection, including reduced serum proinflammatory cytokines storm (eg., TNF-, IL-15, IL-22, G-CSF, M-CSF, MCP-1) and restored amino acid and energy metabolism (Palermo et al., 2023)