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The anxiolytic effects are comparable to benzodiazepines, but without the addiction potential, sedation, or cognitive blunting that makes benzodiazepines problematic for long-term use

On the other hand, there is evidence that spinal cord injury triggers a cascade of secondary degenerative events that cause further damage to the injured area and induce local inflammation along with hemorrhage and edema [12, 13] and that the therapeutic agents imatinib (which has been shown to inhibit cytokine production and reduce hemorrhage, edema, and inflammation) [14] and ibuprofen initiate favorable axonal growth and functional recovery through Rho inhibition [15]

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Tightly coupled repetitive blast-induced traumatic brain injury: development and characterization in mice

benefits build over 28 weeks