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Therefore, we proposed that PD-1 inhibitor might influence CFs through its effects on other cardiac cells via cell-cell crosstalk

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The patient had compound heterozygous nonsense (R387X) and missense (V393A) mutations in the COQ2 gene

Instead, it indicates that cellular glutathione might function as a net reductant of the ER, and might protect the ER against transient hyperoxidizing conditions