glutathione mtorc1 Molecular pathways governing the regulation of ferroptosis. Four mTOR signaling networks: mechanistic insights

Similar content being viewed by others Main Cells that undergo senescence stably exit the cell cycle and display distinct phenotypic changes, which include the production of a bioactive secretome (the senescence-associated secretory phenotype (SASP)), reprogramming their metabolism and becoming more resistant to cell death 1

Altered neurometabolite levels, including glutathione (GSH) and gamma-aminobutyric acid (GABA), have been proposed as potential contributors to the biology underlying ASD

2021;85(6):e413

Intergroup comparisons were performed by parametric t test and ANOVA, or 2 tests

Partitioning heritability by functional annotation using genome-wide association summary statistics