glutathione persister cell Additional data demonstrating that cells have a disabled Sublethal cytochrome c release generates

doi: 10.1007/s00424-003-1192-y 155 BhutiaYDGanapathyV

While selenium deficiency is a major etiological factor of Keshan disease (54), the seasonal and annual variation in disease occurrence suggested that other factors, especially an infectious agent, might be involved in addition to selenium deficiency (55)

coli , as a periplasmic protein along with glutathione as the most abundant thiol (Fahey et al., 1978

However, in hypoxic-ischemic encephalopathy, LCN2 induces Mitochondrial Dysfunction and promotes ferroptosis in HT22 cells (mouse hippocampal neurons) by activating the NF-B/STAT3 signaling axis (Luo et al

[DOI] [PubMed] [Google Scholar] [93].Alavian KN, Beutner G, Lazrove E, Sacchetti S, Park HA, Licznerski P, Li H, Nabili P, Hockensmith K, Graham M, Porter GA, Jr, Jonas EA