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The absence of smad4 was also found to enhance mitochondrial ATP synthase inhibitory factor (ATPIF1) activity, ATP synthase inhibition, and reduce mitochondrial membrane potential, ultimately promoting glycolysis by decreasing glucose metabolic flux and increasing glucose accumulation in the podocyte [62]

In: HurleyWL, editor

Tumor redox heterogeneity, variations in intracellular GSH levels, and discrepancies between in vitro and in vivo redox environments make precise prediction of therapeutic activation difficult

This was largely due to concerns about the methodological quality of the included studies and imprecision in the pooled effect estimates

Gene and protein characterisation of the human glutathione S-transferase kappa and evidence for a peroxisomal localisation