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Future studies are aimed at dissecting APP-dependent mechanisms of tau pathogenesis in patient-derived neurons

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In adolescents and adults of both genders, diagnosed with NASH or hepatic steatosis, CD36 gene and protein expression were increased compared to healthy controls

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This chronic redox imbalance interferes with mitochondrial biogenesis, ATP synthesis, and redox homeostasis, thereby impairing follicular development and oocyte quality (Yan et al., 2024)

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