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A., Carrillo-Garcia, J., Tuntithavornwat, S., et al

Dysregulated neuroimmune signaling, including the classical complement cascade, further contributes to synaptic dysfunction, and blocking this pathway can protect synapses and memory, highlighting the microgliasynapse axis as a potential therapeutic target (Zhong L

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447 Furthermore, astrocytic atrophy and reduced presence of astrocytes around synapses affects synaptic maintenance, glutamate homoeostasis, and K + buffering thus affecting neuronal excitability and limiting synaptic plasticity