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The predicted bioactivation pathway was formation of an aldehyde from the piperidine moiety ( 3 ) was designed to reduce the bioactivation potential

Letrozole-treated boys with ISS showed no loss of bone density [92, 93], but some mild vertebral deformities were observed in prepubertal boys with ISS or a delayed onset of puberty treated with letrozole for 2 years [71, 93]

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Approaches that combine several EV protein measures may be the most fruitful for the clinic and for trials, as these significantly increase AUCs for separating PD from other proteinopathies (to 0.98) 106 , and some studies suggest potential for meaningful quantification 108