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While low dose endotoxin triggered NAMPT-dependent NAD + salvage pathway to support pro-inflammatory response, high dose endotoxin drove a switch from NAD + salvage to IDO1-dependent NAD + de novo biosynthesis at late stage, leading to persistent anti-inflammatory properties and immune tolerance [98]

Coordinated regulation of ppar Expression and activity through control of chromatin structure in adipogenesis and obesity

Cell fraction was collected and processed as described above

Local alignment of ligand binding sites in proteins for polypharmacology and drug repositioning

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