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We observed significant plaque deposition in AL-fed females which was significantly diminished by both DL and CR, as shown by the reduced plaque area in both the cortex and hippocampus of female 3xTg mice fed these diets (Fig

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6,20 Infants with G6PD deficiency and a mutation of uridine diphosphoglucuronate glucuronosyltransferase-1 gene promoter (UDPGT-1) are particularly susceptible to hyperbilirubinemia secondary to decreased liver clearance of bilirubin

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This effect is attributed to the upregulation of hepatic -catenin, which helps to restore the integrity of tight junctions and normalize the expression of bile transport proteins (OATP2, BSEP, and NTCP1), ultimately inhibiting intrahepatic bile duct dilation, cholestasis, and the progression of fibrosis