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postulated that individuals with lower GPx activity are predisposed to impaired antioxidant protection, which leads to oxidative damage to membrane fatty acid s and functional proteins (Fig

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These experiments reveal the multi-faceted impact of RSL3-mediated inhibition of GPX4 in thyroid cancer cells, by altering DNA damage repair and proliferation signaling pathways, and suppressing thyroid tumor cell growth

suggest that the improvement in insulin parameters could be due to the inhibition of COX-2 expression, P-selectin, or inflammatory cytokines such as TNF- and IL-1

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