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Molecular characterization of the family of N-methyl-D-aspartate receptor subunits

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A high level of HLA-I on MM cell membranes may be involved in protecting these cells from NK-mediated attack and contribute to the immune evasion [39]

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[9] Coppers toxicity mechanism is well characterised: free or loosely bound Cu(II)/Cu(I) participates in Fenton-type redox chemistry, generating hydroxyl radicals that damage lipids, proteins, and DNA