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2017), these excitotoxicity-driven mechanisms ultimately lead to cognitive decline, epilepsy, and neurological deficits

CRC Press, Boca Raton, pp 581595 Glassford JAG (2017) The neuroinflammatory etiopathology of myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS)

L'valuation des fonctions neuromusculaires peut tre ralise l'aide de mthodes lectrophysiologiques : mesure de l'excitabilit neuromusculaire (LABORIT H

With respect to VHE, Verrotti and coworkers [69] demonstrated an increase in glutamine production in astrocytes whereas glutamine release was inhibited