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Conversely, the overexpression of Parkin resulted in a reduction in the number of neurons lost in the motor cortex of TDP-43 model animals while also inhibiting the degenerative phenotype observed in hTDP-43 model animals ( It has been demonstrated that excessive mitochondrial fission or fragmentation is a factor in the pathology of amyotrophic lateral sclerosis (ALS)

Counts below this indicate thrombocytopenia

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