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Furthermore, a reduction in cortical parvalbumin-positive interneurons, a hallmark of SZ, can also be triggered by NMDAR antagonists 14 , suggesting that they may be particularly vulnerable to NMDAR hypofunction (the relatively young age of the rats used in the current study prevented analysis of parvalbumin-positive interneurons since they are yet to develop)

monocytogenes controlled its biphasic life cycle and transitioned from saprophytic to pathogenic by regulating the concentrations of GSH (Reniere et al., 2015)

115 This finding reinforces the role of DNA methylation profiling in optimizing treatment regimens

When sulforaphane enters the cell, it interacts with Keap1, causing it to release Nrf2

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