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The hepatotoxicity of chloroform was reported to be due to phosphogene-mediated cellular glutathione depletion or increased amounts of covalent binding to hepatocellular macromolecules [195,196]

Journal of Clinical Biochemistry and Nutrition, 48 (1), 26-32

R., Deng W., Selvaraj V

Relative to PBO F3 progressors, PGZ-treated progressors demonstrated improvement in histological disease activity and multiple liver-related NITs, indicating that PGZ may have clinical benefits even in patients with histological fibrosis progression