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So, for correct human cardiac function, there is a fine balance between excess inhibition of SERCA2a by PLN and no inhibition

induced more iron accumulation and a stronger inflammatory response in STEAP3-knockout mice through lipopolysaccharide (LPS) induction, which may be due to the induction of cellular ferroptosis 52

Second, leveraging the MicroecologyPharmacokinetics Interaction framework, integrate microbial changes, key metabolite exposure (AUC/Cmax), and therapeutic outcomes into a single analytical system

16 , dmm.050231 (2023)

ferroptosis cannot be explained by a simple increase in H 2 O 2 -dependent, iron-catalyzed ROS production (i.e