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Cardiac-specific overexpression of acyl-CoA synthetase-1 (ACSL1) in mice has been shown to mitigate pressure overload-induced cardiac dysfunction by reducing toxic lipid accumulation, restoring long-chain acyl-CoA levels, and enhancing mitochondrial bioenergetics

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We detected a significant reduction of the dehydrogenase activity after incubation with all tested compounds at the highest concentrations

Remember to also watch what you eat, drink more water and get more beauty sleep