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Emerging evidence suggests that early-stage AD patients are also at risk for epilepsy, and this proportion is not low (237, 238)

Recently, several peroxisomal proteins, such as LonP2, insulin-degrading enzyme, and PEX11, have been suggested to contribute to the maintenance of peroxisomal redox homeostasis, similar to the abovementioned antioxidants 34,35,36

In vivo relationship between horny layer reservoir effect and percutaneous absorption in human and rat

Dysfunctional endothelial nitric oxide synthase can further promote ROS production, which exacerbates endothelial damage by reducing NO synthesis and secretion