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presented a magnetically targeted nanocarrier loaded with the PPAR agonist 15d-PGJ2-MNPs, which, when administered intravenously, activated PPAR receptors on macrophages around haematomas, attenuated brain damage, and improved sensory and motor functions in mice after ICH (Wang et al., 2023)

While any oxidation of the GSH is to be avoided, the GSSG has less toxicity than ROS-products of GSH, and indeed GSSG is known to be absorbed and available for hepatic reduction to GSH, though at the cost of increased metabolic load with respect to GSH administration

D., Vinci, D., & Haff, G

Retrieved 13 February 2026