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In this kind of apoptosis, Bax activation and translocation leads to status alterations of mitochondrial permeability transition pores, facilitating depolarization of mitochondrial membrane potential and subsequent release of cytochrome c and other apoptosis-promoting proteins to cytosol (Kim et al., 2021

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Reznick AZ, Packer L (1994) Oxidative damage to proteins: spectrophotometric method for carbonyl assay

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