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UA treatment (16 mg/kg, intravenously) immediately after transient ischemia results in a consistent reduction in brain damage and neurological impairment [148]

Notably, we found age-dependent brain atrophy in GCLC floxed X CaMKII-Cre mice (GCLC-KO) mice but not in wild-type (WT) mice (Fig

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Early-life LPS exposure induce microglial activation and triggers persistent DA neuron loss from infancy through adulthood, with maximal vulnerability observed at embryonic day 10.5a critical developmental window for DA neurogenesis (Zetterstrm et al., 1996)

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