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Incapacitation of any element of the E-cadherin-NF2-Hippo pathway contributes to the expression or activity of its downstream molecule YAP, leaving cancer cells or mutant tumors like NF2- mutant mesotheliomas especially vulnerable to FINs [59, 109]

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& Varner, D

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Cancer cells exploit this paradox by modulating their redox state to support survival, metastasis, and therapeutic resistance

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Development and validation of a novel plasma metabolomic signature for the detection of RCC

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