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For example, eight Australian families with Lebanese ancestry diagnosed with LS or LS-like encephalopathy associated with COX deficiency had mutations in the PET100 gene (Lim et al., 2014)

Cell death by ferroptosis is genetically prevented by the lipid repair enzyme glutathione peroxidase 4 (GPX4), which converts lipid hydroperoxides to unreactive lipid alcohols [17]

(1988), supra)

[74] found that FA-binding protein 5 interacts with FASN, positively regulating its expression through the ubiquitinproteasome pathway, which activates the Wnt/-catenin signaling pathway and promotes lipid metabolism, playing a critical role in the progression of pancreatic neuroendocrine tumors