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indicated that elesclomol could chelate extracellular copper at very low concentrations (nM levels) and enter cells in the form of elesclomol-Cu 2+ complex, selectively targeting the mitochondria and the mitochondrial enzyme FDX1

Varying organ types throughout the month also ensures a balanced nutrient intake

It is studied for its potential in tissue remodeling and cellular response pathways without systemic effects

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Overall, our research on GSH-responsive AP probe-net affords a new strategy for targeted chemotherapy in ATC