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2014;9(5):e96755

The loss of protection during menopause suggests that E1, the predominant postmenopausal oestrogen, exerts limited metabolic effects compared with E2

10.1021/acs.macromol.1c00649 4 BurkeH

In addition to the response to infection, tissue damage itself can initiate inflammation, through the release of damage-associated molecules including arachadonic acid metabolites, free extracellular matrix molecules, nucleotides and others (Van Crombruggen et al., 2013)