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Yes, but only to an extent, and sometimes it takes strategic clinical support at the cellular level

injection, as described previously 11

The increase in serum AST and ALT due to AZA causes inflammation, free radical damage, and cell damage that changes the permeability of hepatocyte membranes, leading to the release of AST and ALT into the bloodstream (61)

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Obviously this ignores all those who require life long treatment due to other causes of B12 deficiency (of which there are many), thereby potentially causing great harm to the vast majority of their B12 deficient patients