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The carboxy-terminal Neh3 Domain of Nrf2 Is required for transcriptional activation

Da c nhiu np nhn vi nhiu chm i mi hn trc

J.MallardB.et al

This mitochondrial fission triggers an upregulation of CHAC1/BOTCH through the transcription factor NRF2 and ROS signaling, which in turn inhibits stem cell self-renewal and promotes cell differentiation ( 2.4.1 The antagonistic roles of CHAC1 and CHAC2 in stem cell regulation The function of CHAC1 is further refined by its competitive interaction with its homolog, CHAC2

Aflatoxin exposure has been implicated in disrupting glucose metabolism and insulin sensitivity, potentially contributing to the development of metabolic syndrome and type 2 diabetes mellitus