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Importantly, elevated PI3K/Akt activity has been observed in IPF lung tissues and in bleomycin-induced pulmonary fibrosis models, underscoring its pathophysiological relevance (Wang et al., 2022a)

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These experiments identified a conserved pattern of lipid depletion that progressed from the utilization of metabolically responsive adipocytes to catabolism of stable adipocytes, mirroring outcomes in end-stage starvation, cachexia and severe anorexia

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