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Among the hallmarks of senescence we find metabolic alterations, including an increase in reactive oxygen species formation at the mitochondrial level (Suryadevara et al., 2024), that participates in the induction and maintenance of the senescent phenotype (Lee et al., 1999

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The repeated NAD + /NADH conversions also suggest much more complicated and repeated oxidation/reduction responses in brain cortex during neuronal and astrocytic activities than previously considered (e.g., Kasischke et al., over 80% of neuronal oxidative ATP production is coupled to neuronal signaling even in the absence of specific stimulation (Rothman et al., + /NADH conversions occur in astrocytes