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MicroRNA-34a and MicroRNA-181a mediate visfatin-induced apoptosis and oxidative stress via NF-B pathway in human osteoarthritic chondrocytes

One possibility is that AMPK potentiates, albeit indirectly, the GAP activity of TSC2 towards Rheb, converting Rheb to its inactive, GDP-bound form that causes reductions in cell size and enhanced cell survival in the face of glucose deprivation [24]