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In the progression of liver cancer, c-Met is regulated by various factors such as miRNAs and SOCS1

The immunology of stroke: from mechanisms to translation

The sooner you address the underlying tissue damage, the better your chances of achieving complete healing and avoiding invasive procedures

Treatment with PHA665752, a second generation of the highly selective c-MET inhibitor, causes inactivation of c-MET, leading to the inactivation of AKT and downstream signaling of molecules FOXO1 and GSK3 in DLBCL cell lines