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Kaleli S

They found that after DCG066 intervention, the levels of ROS, iron, and MDA in MM cells increased significantly, while the level of GSH decreased

Excessive production of ECM components, coupled with chronic inflammation, promotes the progression from steatosis to hepatic fibrosis, cirrhosis, and ultimately liver failure (90)

On the other hand, a tyrosine kinase inhibitor, called nintedanib, primarily targets VEGF, FGF, and PDGF receptors and reduces ECM deposition and fibroblast proliferation [9, 14]

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