glutathione and triple negative breast cancer Epithelial, Mesenchymal, Hybrid triple-negative Activation of the eIF2α/ATF4 axis

Activation of the eIF2 ATF4 axis drives triple negative breast cancer radioresistance by promoting glutathione biosynthesis ScienceDirect Metabolic interplay between exogenous cystine and glutamine dependence in triple negative breast cancer Cell Death Discovery cd44 glutathione Receptor Mediated Ferroptosis Induction by Hyaluronic Acid Carbon Quantum Dots in Triple Negative Breast Cancer Cells Through Downregulation of SLC7A11 Pathway Reduction sensitive CD44 receptor targeted hyaluronic acid Glutathione depleting self immolative nanoparticles boost cuproptosis driven metalloimmunotherapy for triple negative breast cancer Journal of Nanobiotechnology Springer Nature Link Mitochondrial Redox Vulnerabilities in Triple Negative Breast Cancer: Integrative Perspectives and Emerging Therapeutic Strategies

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