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There have also been new findings regarding targeted therapy against C

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In vivo studies suggested it to be resulting from oscillations of inhibition and excitatory thalamic and corticothalamic neurons, which in turn, initiate Grand Mal epilepsy [80]

The concept is gaining traction as more individuals seek alternative therapies to alleviate inflammation

The main data supporting the results in this study are available within the paper and its Supplementary Information