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Freed, Rachel D., et al

2024 Jul 15];74

Another in vitro EAAT2-inducing compound, MS-153, effectively protected against secondary neurodegeneration after traumatic brain injury, but through mechanisms other that EAAT2 upregulation (Fontana et al., As described in the Section on The Concepts of Acute and Chronic Glutamate Toxicity, Glud1 Tg mice represent a model of chronic excitotoxicity mediated by increased synaptic L-glu release with limited neuronal loss (Bao et al., 1997

Besides that, it has been shown that heme-IgG complex can interact with previously unrecognized bacterial antigens and intact bacteria through binding to an enlarged panel of structurally unrelated epitopes (146)