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This dual block on NF-B and MAPK suppresses production of IL-8, IL-6, IL-12, IFN-, IL-1, and TNF-

Research shows NNMT inhibition dramatically improves how cells produce and utilize energy

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By specifically binding to cardiolipin to form a stable complex, it not only enhances the damage resistance of the mitochondrial membrane, effectively defending against the invasion of -amyloid toxic oligomers, but also optimizes the efficiency of the electron transport chain, reducing the generation of reactive oxygen species at the source.Concurrently, it upregulates the activity of endogenous antioxidant enzymes, alleviates oxidative damage to neuronal membranes and organelles, significantly mitigates the neurotoxicity of -amyloid, and inhibits further expansion of fibrillar plaques