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Given the established evidence of platelet involvement in breast cancer and other cancer metastases, there has been interest in using antiplatelet therapies to manage cancer progression

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and M2 macrophages (M2) uses the effective coupling of respiration in the electron transport chain to enhance oxidative phosphorylation (OXPHOS), reduces ROS production, and promotes improved obesity and systemic glucose homeostasis by inhibiting adipocytes proliferation (136, 137)