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4.3 Fatty acid metabolism: synthesis, oxidation, and lipid reprogramming Fatty acid metabolic reprogramming in colorectal cancer constitutes a fundamental metabolic pillar supporting malignant phenotypes

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Disrupting HDAC4-Ubc9 interactions restores homologous recombination proficiency in hepatocellular carcinoma [72], while targeting USP38-HDAC1 stabilization compromises NHEJ efficiency by suppressing chromatin compaction [6]

The WIPO platform has also been used, inserting as keywords N-acetylcysteine, and transdermal administration. The authors examined the resulting lists of abstracts, excluding those that did not fit within the scope of the present work