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Diabet Med 10 : 833838 Press M et al

Given the robustness of this mitochondrial response, the multifaceted nature of PGC1 regulation in cells and a modest rescue at best by mTORC1 inhibition, we suspect that there is a degree of functional redundancy built into this metabolic plasticity system that can contribute to such an adaptive response, and complete inhibition of this system will prove difficult

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Beyond its role in energy metabolism, NAD+ is integral to DNA repair, cellular signaling, and the regulation of various biological processes, including aging and inflammation