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Loss of GATOR1 function has been shown to augment glucose starvation-induced phosphorylation of Raptor by AMPK, but not the classical AMPK substrate acetyl-CoA carboxylase (ACC), despite hyperactivity of mTORC1 [55]

But that presents somewhat of a Catch 22

Rather than focusing on catalog size, we prioritized transparency, traceability, and the availability of supporting quality records

Krishnamurthy et al., 2024)