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One possible explanation, however, is that cysteine levels are elevated in the HepG2/C3A cells as part of a protective adaptation against the much higher oxygen tensions associated with cell culture systems

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Collectively, these findings suggest that the antioxidant NAC treatment successfully rescued the exacerbated ROS generation, SGNs loss, and apoptosis caused by GPX1-deficiency after peroxynitrite damage

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Studies suggest that IV antioxidant therapy can help manage neurofunctional pain by correcting deficiencies and reducing inflammation in the nervous system