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1D), which was consistent with previous studies suggesting that the mRNA expression levels of Ppara were inhibited after APAP treatment in WT mice [15, 16]

D-galactose induces damage to hippocampal neurons by increasing ROS production and advanced glycation end product accumulation, leading to oxidative stress, mitochondrial dysfunction, and neuroinflammation

Dex modulates the balance of water-electrolyte metabolism by depressing the expression of AVP in PVN

Hypoxia depresses CD8 + T cell functions in HCC Hypoxia is a critical characteristic of the TME in solid tumours, including HCC, and plays a key role in creating an immunosuppressive TME

Through the in vitro experiments, the inhibition of mTORC1/HIF-1 pathway is responsible for the reduced glycolysis observed under dioscin treatment