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Energy metabolism in ALS: An underappreciated opportunity

Tu YXI, Sydor AM, Coyaud E, Laurent EMN, Dyer D, Mellouk N, et al

Funding This work was supported in part by the Al Jalila Foundation

For instance, tumors with high endogenous CCNE1 expression, which encodes the oncogene cyclin E1, are HR-proficient and inherently resistant to DNA damaging agents (Patch et al., 2015)

For essential metabolic enzymes that are abundantly expressed, even 9095% knockdown does not lead to significant growth defects, and 510% of enzyme activity from the leftover protein seems to be adequate for normal parasite growth 62