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This review consolidates existing evidence to support a key argument: mitochondrial dysfunction serves as a common pathogenic mechanism in neurodegenerative diseases, linking various pathological features into a cohesive framework

In the second step, glycine is added to the gamma-glutamylcysteine using the enzyme glutathione synthetase to form glutathione

When cancerous cells are generated, the expression of GPx1 is abnormal, causing intracellular ROS dysfunction [77, 78]

Diagnosis and management of hemochromatosis