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When the mTORC1 pathway was inhibited by rapamycin treatment after ATF4 knockdown, the significant differences in autophagy levels disappeared between the shATF4 and shNC groups ( Figure 8D )

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Psychological disorders such as Alzheimer's disease have been shown to cause oxidative damage to glycolytic enzymes as well as enzymes involved in the tricarboxylic cycle during glucose metabolism, thereby affective ATP biogenesis (169)

Suppression of SHP-2 and ERK signalling promotes self-renewal of mouse embryonic stem cells

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